Wolff-Parkinson-White syndrome
Identifieur interne : 000134 ( Main/Corpus ); précédent : 000133; suivant : 000135Wolff-Parkinson-White syndrome
Auteurs : Andrew G. Wallace ; John P. Boineau ; Robert M. Davidson ; Will C. SealySource :
- The American Journal of Cardiology [ 0002-9149 ] ; 1971.
Abstract
In the past 4 years physiologic studies of patients with the Wolff-Parkinson-White (WPW) syndrome and its variants have shed new light on the structural basis for this electrocardiographic abnormality and the mechanisms responsible for supraventricular arrhythmias. Accessory pathways for atrioventricular propagation appear to be responsible for the electrocardiographic abnormality in the majority of the patients with this syndrome. These pathways may be similar to those described by Kent and located in the lateral portion of either the right or left atrioventricular groove, or the accessory bundles bypassing the atrioventricular node may be located within the septal region. Careful analysis of the electrocardiogram, atrial pacing studies and the records from the bundle of His can be used to identify the probable location of the pathway as well as certain of its functional characteristics. Surgical interruption of the accessory pathway provides a definitive form of therapy, although this approach is not necessary in the majority of patients with WPW syndrome. Furthermore, surgical intervention should be entertained only after physiologic studies have been made and in a setting in which careful epicardial mapping can be performed at the time of surgical intervention.
Url:
DOI: 10.1016/0002-9149(71)90092-0
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<record><TEI wicri:istexFullTextTei="biblStruct"><teiHeader><fileDesc><titleStmt><title xml:lang="en">Wolff-Parkinson-White syndrome</title><author><name sortKey="Wallace, Andrew G" sort="Wallace, Andrew G" uniqKey="Wallace A" first="Andrew G." last="Wallace">Andrew G. Wallace</name><affiliation><mods:affiliation>From the Departments of Medicine and Surgery, Duke University Medical Center, Durham, N. C. USA</mods:affiliation></affiliation></author><author><name sortKey="Boineau, John P" sort="Boineau, John P" uniqKey="Boineau J" first="John P." last="Boineau">John P. Boineau</name><affiliation><mods:affiliation>From the Departments of Medicine and Surgery, Duke University Medical Center, Durham, N. C. USA</mods:affiliation></affiliation></author><author><name sortKey="Davidson, Robert M" sort="Davidson, Robert M" uniqKey="Davidson R" first="Robert M." last="Davidson">Robert M. Davidson</name><affiliation><mods:affiliation>From the Departments of Medicine and Surgery, Duke University Medical Center, Durham, N. C. USA</mods:affiliation></affiliation></author><author><name sortKey="Sealy, Will C" sort="Sealy, Will C" uniqKey="Sealy W" first="Will C." last="Sealy">Will C. Sealy</name><affiliation><mods:affiliation>From the Departments of Medicine and Surgery, Duke University Medical Center, Durham, N. C. USA</mods:affiliation></affiliation></author></titleStmt><publicationStmt><idno type="wicri:source">ISTEX</idno><idno type="RBID">ISTEX:7715B38D1492FDC0A00D811C31786015306DF2F1</idno><date when="1971" year="1971">1971</date><idno type="doi">10.1016/0002-9149(71)90092-0</idno><idno type="url">https://api.istex.fr/document/7715B38D1492FDC0A00D811C31786015306DF2F1/fulltext/pdf</idno><idno type="wicri:Area/Main/Corpus">000134</idno></publicationStmt><sourceDesc><biblStruct><analytic><title level="a" type="main" xml:lang="en">Wolff-Parkinson-White syndrome</title><author><name sortKey="Wallace, Andrew G" sort="Wallace, Andrew G" uniqKey="Wallace A" first="Andrew G." last="Wallace">Andrew G. Wallace</name><affiliation><mods:affiliation>From the Departments of Medicine and Surgery, Duke University Medical Center, Durham, N. C. USA</mods:affiliation></affiliation></author><author><name sortKey="Boineau, John P" sort="Boineau, John P" uniqKey="Boineau J" first="John P." last="Boineau">John P. Boineau</name><affiliation><mods:affiliation>From the Departments of Medicine and Surgery, Duke University Medical Center, Durham, N. C. USA</mods:affiliation></affiliation></author><author><name sortKey="Davidson, Robert M" sort="Davidson, Robert M" uniqKey="Davidson R" first="Robert M." last="Davidson">Robert M. Davidson</name><affiliation><mods:affiliation>From the Departments of Medicine and Surgery, Duke University Medical Center, Durham, N. C. USA</mods:affiliation></affiliation></author><author><name sortKey="Sealy, Will C" sort="Sealy, Will C" uniqKey="Sealy W" first="Will C." last="Sealy">Will C. Sealy</name><affiliation><mods:affiliation>From the Departments of Medicine and Surgery, Duke University Medical Center, Durham, N. C. USA</mods:affiliation></affiliation></author></analytic><monogr></monogr><series><title level="j">The American Journal of Cardiology</title><title level="j" type="abbrev">AJC</title><idno type="ISSN">0002-9149</idno><imprint><publisher>ELSEVIER</publisher><date type="published" when="1971">1971</date><biblScope unit="volume">28</biblScope><biblScope unit="issue">5</biblScope><biblScope unit="page" from="509">509</biblScope><biblScope unit="page" to="515">515</biblScope></imprint><idno type="ISSN">0002-9149</idno></series><idno type="istex">7715B38D1492FDC0A00D811C31786015306DF2F1</idno><idno type="DOI">10.1016/0002-9149(71)90092-0</idno><idno type="PII">0002-9149(71)90092-0</idno><idno type="ArticleID">71900920</idno></biblStruct></sourceDesc><seriesStmt><idno type="ISSN">0002-9149</idno></seriesStmt></fileDesc><profileDesc><textClass></textClass><langUsage><language ident="en">en</language></langUsage></profileDesc></teiHeader><front><div type="abstract" xml:lang="en">In the past 4 years physiologic studies of patients with the Wolff-Parkinson-White (WPW) syndrome and its variants have shed new light on the structural basis for this electrocardiographic abnormality and the mechanisms responsible for supraventricular arrhythmias. Accessory pathways for atrioventricular propagation appear to be responsible for the electrocardiographic abnormality in the majority of the patients with this syndrome. These pathways may be similar to those described by Kent and located in the lateral portion of either the right or left atrioventricular groove, or the accessory bundles bypassing the atrioventricular node may be located within the septal region. Careful analysis of the electrocardiogram, atrial pacing studies and the records from the bundle of His can be used to identify the probable location of the pathway as well as certain of its functional characteristics. Surgical interruption of the accessory pathway provides a definitive form of therapy, although this approach is not necessary in the majority of patients with WPW syndrome. Furthermore, surgical intervention should be entertained only after physiologic studies have been made and in a setting in which careful epicardial mapping can be performed at the time of surgical intervention.</div></front></TEI><istex><corpusName>elsevier</corpusName><author><json:item><name>Andrew G. Wallace MD</name><affiliations><json:string>From the Departments of Medicine and Surgery, Duke University Medical Center, Durham, N. C. USA</json:string></affiliations></json:item><json:item><name>John P. Boineau MD</name><affiliations><json:string>From the Departments of Medicine and Surgery, Duke University Medical Center, Durham, N. C. USA</json:string></affiliations></json:item><json:item><name>Robert M. Davidson MD</name><affiliations><json:string>From the Departments of Medicine and Surgery, Duke University Medical Center, Durham, N. C. USA</json:string></affiliations></json:item><json:item><name>Will C. Sealy MD</name><affiliations><json:string>From the Departments of Medicine and Surgery, Duke University Medical Center, Durham, N. C. USA</json:string></affiliations></json:item></author><articleId><json:string>71900920</json:string></articleId><language><json:string>eng</json:string></language><abstract>In the past 4 years physiologic studies of patients with the Wolff-Parkinson-White (WPW) syndrome and its variants have shed new light on the structural basis for this electrocardiographic abnormality and the mechanisms responsible for supraventricular arrhythmias. Accessory pathways for atrioventricular propagation appear to be responsible for the electrocardiographic abnormality in the majority of the patients with this syndrome. These pathways may be similar to those described by Kent and located in the lateral portion of either the right or left atrioventricular groove, or the accessory bundles bypassing the atrioventricular node may be located within the septal region. Careful analysis of the electrocardiogram, atrial pacing studies and the records from the bundle of His can be used to identify the probable location of the pathway as well as certain of its functional characteristics. Surgical interruption of the accessory pathway provides a definitive form of therapy, although this approach is not necessary in the majority of patients with WPW syndrome. 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S. Public Health Service.</note><note type="content">Section title: Symposium on Electophysiologic Correlates of Clinical Arrhythmias. Part III</note></notesStmt><sourceDesc><biblStruct type="inbook"><analytic><title level="a" type="main" xml:lang="en">Wolff-Parkinson-White syndrome</title><title level="a" type="sub" xml:lang="en">A new look</title><author><persName><forename type="first">Andrew G.</forename><surname>Wallace</surname></persName><roleName type="degree">MD</roleName><note type="biography">Address for reprints: Andrew G. Wallace, MD, Box 3084, Duke University Medical Center, Durham, N. C. 27706.</note><affiliation>Address for reprints: Andrew G. Wallace, MD, Box 3084, Duke University Medical Center, Durham, N. C. 27706.</affiliation><affiliation>From the Departments of Medicine and Surgery, Duke University Medical Center, Durham, N. C. USA</affiliation></author><author><persName><forename type="first">John P.</forename><surname>Boineau</surname></persName><roleName type="degree">MD</roleName><affiliation>From the Departments of Medicine and Surgery, Duke University Medical Center, Durham, N. C. USA</affiliation></author><author><persName><forename type="first">Robert M.</forename><surname>Davidson</surname></persName><roleName type="degree">MD</roleName><affiliation>From the Departments of Medicine and Surgery, Duke University Medical Center, Durham, N. C. USA</affiliation></author><author><persName><forename type="first">Will C.</forename><surname>Sealy</surname></persName><roleName type="degree">MD</roleName><affiliation>From the Departments of Medicine and Surgery, Duke University Medical Center, Durham, N. C. USA</affiliation></author></analytic><monogr><title level="j">The American Journal of Cardiology</title><title level="j" type="abbrev">AJC</title><idno type="pISSN">0002-9149</idno><idno type="PII">S0002-9149(00)X0478-X</idno><imprint><publisher>ELSEVIER</publisher><date type="published" when="1971"></date><biblScope unit="volume">28</biblScope><biblScope unit="issue">5</biblScope><biblScope unit="page" from="509">509</biblScope><biblScope unit="page" to="515">515</biblScope></imprint></monogr><idno type="istex">7715B38D1492FDC0A00D811C31786015306DF2F1</idno><idno type="DOI">10.1016/0002-9149(71)90092-0</idno><idno type="PII">0002-9149(71)90092-0</idno><idno type="ArticleID">71900920</idno></biblStruct></sourceDesc></fileDesc><profileDesc><creation><date>1971</date></creation><langUsage><language ident="en">en</language></langUsage><abstract xml:lang="en"><p>In the past 4 years physiologic studies of patients with the Wolff-Parkinson-White (WPW) syndrome and its variants have shed new light on the structural basis for this electrocardiographic abnormality and the mechanisms responsible for supraventricular arrhythmias. Accessory pathways for atrioventricular propagation appear to be responsible for the electrocardiographic abnormality in the majority of the patients with this syndrome. These pathways may be similar to those described by Kent and located in the lateral portion of either the right or left atrioventricular groove, or the accessory bundles bypassing the atrioventricular node may be located within the septal region. Careful analysis of the electrocardiogram, atrial pacing studies and the records from the bundle of His can be used to identify the probable location of the pathway as well as certain of its functional characteristics. Surgical interruption of the accessory pathway provides a definitive form of therapy, although this approach is not necessary in the majority of patients with WPW syndrome. Furthermore, surgical intervention should be entertained only after physiologic studies have been made and in a setting in which careful epicardial mapping can be performed at the time of surgical intervention.</p></abstract></profileDesc><revisionDesc><change when="1971">Published</change></revisionDesc></teiHeader></istex:fulltextTEI></fulltext><metadata><istex:metadataXml wicri:clean="Elsevier, elements deleted: tail"><istex:xmlDeclaration>version="1.0" encoding="UTF-8"</istex:xmlDeclaration><istex:docType PUBLIC="-//ES//DTD journal article DTD version 4.5.2//EN//XML" URI="art452.dtd" name="istex:docType"></istex:docType><istex:document><converted-article version="4.5.2" docsubtype="fla" xml:lang="en"><item-info><jid>AJC</jid><aid>71900920</aid><ce:pii>0002-9149(71)90092-0</ce:pii><ce:doi>10.1016/0002-9149(71)90092-0</ce:doi><ce:copyright type="unknown" year="1971"></ce:copyright></item-info><head><ce:article-footnote><ce:label>☆</ce:label><ce:note-para>This work was supported in part by Grants HE 19,949, HE 19,613, HE 05736 and HE 01782 from the U. S. Public Health Service.</ce:note-para></ce:article-footnote><ce:dochead><ce:textfn>Symposium on Electophysiologic Correlates of Clinical Arrhythmias. Part III</ce:textfn></ce:dochead><ce:title>Wolff-Parkinson-White syndrome</ce:title><ce:subtitle>A new look</ce:subtitle><ce:author-group><ce:author><ce:given-name>Andrew G.</ce:given-name><ce:surname>Wallace</ce:surname><ce:degrees>MD</ce:degrees><ce:cross-ref refid="COR1"><ce:sup loc="post">∗</ce:sup></ce:cross-ref></ce:author><ce:author><ce:given-name>John P.</ce:given-name><ce:surname>Boineau</ce:surname><ce:degrees>MD</ce:degrees></ce:author><ce:author><ce:given-name>Robert M.</ce:given-name><ce:surname>Davidson</ce:surname><ce:degrees>MD</ce:degrees></ce:author><ce:author><ce:given-name>Will C.</ce:given-name><ce:surname>Sealy</ce:surname><ce:degrees>MD</ce:degrees></ce:author><ce:affiliation><ce:textfn>From the Departments of Medicine and Surgery, Duke University Medical Center, Durham, N. C. USA</ce:textfn></ce:affiliation><ce:correspondence id="COR1"><ce:label>∗</ce:label><ce:text>Address for reprints: Andrew G. Wallace, MD, Box 3084, Duke University Medical Center, Durham, N. C. 27706.</ce:text></ce:correspondence></ce:author-group><ce:abstract class="author"><ce:section-title>Abstract</ce:section-title><ce:abstract-sec><ce:simple-para view="all" id="simple-para.0010">In the past 4 years physiologic studies of patients with the Wolff-Parkinson-White (WPW) syndrome and its variants have shed new light on the structural basis for this electrocardiographic abnormality and the mechanisms responsible for supraventricular arrhythmias. Accessory pathways for atrioventricular propagation appear to be responsible for the electrocardiographic abnormality in the majority of the patients with this syndrome. These pathways may be similar to those described by Kent and located in the lateral portion of either the right or left atrioventricular groove, or the accessory bundles bypassing the atrioventricular node may be located within the septal region. Careful analysis of the electrocardiogram, atrial pacing studies and the records from the bundle of His can be used to identify the probable location of the pathway as well as certain of its functional characteristics. Surgical interruption of the accessory pathway provides a definitive form of therapy, although this approach is not necessary in the majority of patients with WPW syndrome. Furthermore, surgical intervention should be entertained only after physiologic studies have been made and in a setting in which careful epicardial mapping can be performed at the time of surgical intervention.</ce:simple-para></ce:abstract-sec></ce:abstract></head></converted-article></istex:document></istex:metadataXml><mods version="3.6"><titleInfo lang="en"><title>Wolff-Parkinson-White syndrome</title><subTitle>A new look</subTitle></titleInfo><titleInfo type="alternative" lang="en" contentType="CDATA"><title>Wolff-Parkinson-White syndrome</title><subTitle>A new look</subTitle></titleInfo><name type="personal"><namePart type="given">Andrew G.</namePart><namePart type="family">Wallace</namePart><namePart type="termsOfAddress">MD</namePart><affiliation>From the Departments of Medicine and Surgery, Duke University Medical Center, Durham, N. C. USA</affiliation><description>Address for reprints: Andrew G. Wallace, MD, Box 3084, Duke University Medical Center, Durham, N. 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Accessory pathways for atrioventricular propagation appear to be responsible for the electrocardiographic abnormality in the majority of the patients with this syndrome. These pathways may be similar to those described by Kent and located in the lateral portion of either the right or left atrioventricular groove, or the accessory bundles bypassing the atrioventricular node may be located within the septal region. Careful analysis of the electrocardiogram, atrial pacing studies and the records from the bundle of His can be used to identify the probable location of the pathway as well as certain of its functional characteristics. Surgical interruption of the accessory pathway provides a definitive form of therapy, although this approach is not necessary in the majority of patients with WPW syndrome. Furthermore, surgical intervention should be entertained only after physiologic studies have been made and in a setting in which careful epicardial mapping can be performed at the time of surgical intervention.</abstract><note>This work was supported in part by Grants HE 19,949, HE 19,613, HE 05736 and HE 01782 from the U. S. Public Health Service.</note><note type="content">Section title: Symposium on Electophysiologic Correlates of Clinical Arrhythmias. Part III</note><relatedItem type="host"><titleInfo><title>The American Journal of Cardiology</title></titleInfo><titleInfo type="abbreviated"><title>AJC</title></titleInfo><genre type="Journal">journal</genre><originInfo><dateIssued encoding="w3cdtf">197111</dateIssued></originInfo><identifier type="ISSN">0002-9149</identifier><identifier type="PII">S0002-9149(00)X0478-X</identifier><part><date>197111</date><detail type="issue"><title>Symposium on Electophysiologic Correlates of Clinical Arrhythmias</title></detail><detail type="volume"><number>28</number><caption>vol.</caption></detail><detail type="issue"><number>5</number><caption>no.</caption></detail><extent unit="issue pages"><start>E1</start><end>E54</end></extent><extent unit="issue pages"><start>E59</start><end>E71</end></extent><extent unit="issue pages"><start>499</start><end>620</end></extent><extent unit="pages"><start>509</start><end>515</end></extent></part></relatedItem><identifier type="istex">7715B38D1492FDC0A00D811C31786015306DF2F1</identifier><identifier type="DOI">10.1016/0002-9149(71)90092-0</identifier><identifier type="PII">0002-9149(71)90092-0</identifier><identifier type="ArticleID">71900920</identifier><recordInfo><recordContentSource>ELSEVIER</recordContentSource></recordInfo></mods></metadata><enrichments><istex:refBibTEI uri="https://api.istex.fr/document/7715B38D1492FDC0A00D811C31786015306DF2F1/enrichments/refBib"><teiHeader></teiHeader><text><front></front><body></body><back><listBibl><biblStruct><analytic><title level="a" type="main">References Durrer D, Schuilenburg RM, Wellens HJJ: Pre-excitation revisited</title></analytic><monogr><title level="j">Amer J Cardiol</title><imprint><biblScope unit="volume">25</biblScope><biblScope unit="page" from="69" to="697"></biblScope><date type="published" when="1970"></date></imprint></monogr></biblStruct><biblStruct><analytic><title level="a" type="main">The syndrome of short P-R interval, normal QRS complex and paroxysmal rapid heart action</title><author><persName><forename type="middle">B</forename><surname>Lown</surname></persName></author><author><persName><forename type="middle">Wf</forename><surname>Ganong</surname></persName></author><author><persName><surname>Levine</surname></persName></author><author><persName><surname>Sa</surname></persName></author></analytic><monogr><title level="j">Circulation</title><imprint><biblScope unit="volume">5</biblScope><biblScope unit="page" from="693" to="706"></biblScope><date type="published" when="1952"></date></imprint></monogr></biblStruct><biblStruct><analytic><title level="a" type="main">Bundle branch block with short P-R interval in healthy young people prone to paroxysmal tachycardia</title><author><persName><forename type="middle">L</forename><surname>Wolff</surname></persName></author><author><persName><forename type="middle">J</forename><surname>Parkinson</surname></persName></author><author><persName><surname>White</surname></persName></author></analytic><monogr><title level="j">Amer Heart J</title><imprint><biblScope unit="page" from="53685" to="704"></biblScope><date type="published" when="1930"></date></imprint></monogr></biblStruct><biblStruct><analytic><title level="a" type="main">A case in which the vagus influenced the form of the ventricular complex of the electrocardiogram Hamburger Ww: Bundle branch block. 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